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  • Goat Anti-Mouse IgG2a-AF555

Goat Anti-Mouse IgG2a-AF555

Cat. No.:
1081-32
Goat Anti-Mouse IgG2a-Alexa Fluor® 555 antibody for use in immunohistochemistry / immunocytochemistry and western blot assays.
$249.00
Size Price (USD) Quantity
1.0 mg $249.00
Technical Bulletin Safety Datasheet

Product Specifications

More Information
Isotype Goat IgG
Isotype Control 0109-32
Specificity Reacts with the heavy chain of mouse IgG2a
Source Pooled antisera from goats hyperimmunized with mouse IgG2a
Cross Adsorption Mouse IgG1, IgG2b, IgG3, IgM, and IgA; may react with immunoglobulins from other species
Purification Method Affinity chromatography on mouse IgG2a covalently linked to agarose
Conjugate AF555 (Alexa Fluor® 555)
Buffer Formulation Phosphate buffered saline containing < 0.1% sodium azide
Clonality Polyclonal
Concentration 1.0 mg/mL
Volume 1.0 mL
Recommended Storage 2-8°C; Avoid exposure to light
Trademark Information Alexa Fluor® is a registered trademark of Thermo Fisher Scientific, Inc. or its subsidiaries
Applications Quality tested applications for relevant formats include -
ELISA
FLISA

Other referenced applications for relevant formats include -
Immunohistochemistry-Frozen Sections 1
Immunocytochemistry 1
Western Blot 2
RRID Number AB_2794498
Gene ID 380793 (Mouse)
Gene ID Symbol Ighg2a (Mouse)
Gene ID Aliases 1810060O09Rik; Igh-1; Igh-1a
UniProt ID P01863 (Mouse
P01864 (Mouse
P01865 (Mouse
UniProt Name GCAA_MOUSE (Mouse)
GCAB_MOUSE (Mouse)
GCAM_MOUSE (Mouse)

Documentation

Documentation
Technical Bulletin Safety Datasheet
Certificate of Analysis Lookup

Enter the Catalog Number and Lot Number for the Certificate of Analysis you wish to view


Need help with this product?
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Other Conjugates and Formulations

References and Citations

  1. 1. Piersma B, de Rond S, Werker PM, Boo S, Hinz B, van Beuge MM, et al. YAP1 is a driver of myofibroblast differentiation in normal and diseased fibroblasts. Am J Pathol. 2015;185:3326-37. (IHC-FS, ICC)
  2. 2. Bruns SA. Autophagy regulation by the p38 MAP kinase [dissertation]. Magdeburg (Germany): Otto-von-Guericke-Universität Magdeburg; 2017. (WB)

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